The EMGS Vision
To promote critical scientific knowledge and research into the causes and consequences of damage to the genome and epigenome in order to inform and support national and international efforts to ensure a healthy, sustainable environment for future generations.
Mission of the EMGS
The EMGS mission is (1) to foster scientific research and education on the causes and mechanistic bases of DNA damage and repair, mutagenesis, heritable effects, epigenetic alterations in genome function, and their relevance to disease, and (2) to promote the application and communication of this knowledge to genetic toxicology testing, risk assessment, and regulatory policy-making to protect human health and the environment.
- Provide a broad scientific forum for basic and applied researchers, students and teachers to
- share current scientific research and integrate knowledge on detection and characterization of DNA damage from exposure to man-made or natural toxins in the environment, the molecular mechanisms of DNA repair processes that respond to such damage, and the mechanisms of heritable changes (both mutagenic and epigenetic) that occur when damage persists;
- understand how genomic and environmental factors interact to determine an individual’s susceptibility to adverse health effects and transgenerational defects from such exposure;
- apply this knowledge to assess the risks for adverse health consequences and damage to the biota from environmental toxins.
- Support responsible social and scientific policies by informing the public and by providing recommendations and guidelines to national and international agencies that regulate genome-damaging agents to minimize human and environmental risks.
- Advance basic and applied genome toxicity research by advocating for research support.
- Maintain a diverse membership that fosters and supports scientific interactions among scientists worldwide across academia, government, and industry who are engaged in genome/epigenome toxicity research and its regulatory applications.